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Condoms, jimmies, rubbers…you might think you’ve heard them all. Condoms are one of the world’s most common prophylactics. Here are 5 things that you might not otherwise know about condoms and their names. 1. English Nicknames One of the most known nicknames for condom is rubber. However, if you go to a convenience store in Australia or New Zealand and ask for a rubber, you will be handed an eraser. This could make for awkward moments for Kiwis or Aussies traveling abroad who just want to erase a mistake: “Could I have a rubber, mate?” Other English nicknames include jimmy hat, raincoat, or hazmat suit: a suit you don for dealing with hazardous materials. The term “love glove” led to the famous safe sex slogan “No Glove, No Love.” 2. International Nicknames English isn’t the only language that had odd nicknames for condoms. In Denmark, they are called gummimand, which literally means “rubberman.” In Germany, they are called lummeltute, or “naughty bags.” Hungarian terminology emphasizes the protective aspect by calling a condom an ovsver, or a “safety tool.” Hong Kong similarly demonstrates the protective value by calling a condom a pei dang vi, or a “bulletproof vest.” In Portugal they call condoms “Venus’ shirts” or camisa de Venus: remember, Venus is the goddess of love after all, so it makes sense! Other countries can be more literal with their meanings: in Nigeria, a condom is an okpuamu, or a “penis hat.” In Indonesia, instead of a hat, it’s a “penis gourd” or a koteca. In English a condom is sometimes called a raincoat: in Greek it is sometimes called a kapota, or an overcoat. In Spain, a condom is called a globo, or balloon. Remember, although you can use a condom for a balloon, you can’t use a balloon for a condom! 3. National Tensions Some nicknames of the condoms demonstrate international tensions. In Germany, a slang term for a condom is a “Pariser,” or a Parisian. In English, condoms are sometimes called French Letters. Why is France associated with condoms? This might be because other countries associated all that was decadent with France. As a side note, a French Letter will protect you against the French Disease; or, to put it more plainly, a condom will help protect you against syphilis. Syphilis was called the French Disease because of the outbreak in the French Army in the sixteenth century; it was the Italians that coined that phrase (morbus gallicus). The French, however, might have gotten their linguistic come-uppance with their terminology. The French called syphilis “la maladie anglaise,” or the English Disease. They even called it the Italian disease or the Neapolitan disease too. Other countries were equally derisive, with the Arabs calling syphilis the English disease and the Russians calling it the Polish disease. Although most nationally-derogatory terms for syphilis are now in the past, the French still call condoms “la capote anglaise,” or the English raincoat. 4. Condom, France Yes, there is a town in France called Condom. As far as linguists know, it has nothing do with the etymology of the word condom. There is a folktale that the English got their word condom from this location. English travelers came and saw French farmers sewing prophylactics from sheep guts. Whether or not this is true, you can still get sheepskin condoms (made from sheep intestines). They are softer than latex or polyurethane condoms and increase sensation. However, sheepskin condoms do not protect against sexually transmitted diseases, they simply work to prevent pregnancy. 5. The Real Origins of the Word Condom Unfortunately, we don’t know the real origins of the word “Condom.” Was it named after a Dr. Condom, or a Dr. Quondam, as some tales tell? Is it named after a British army officer, Cundum? Or is it named after the Italian court adviser, Gondi? (A “gondon” or “goldoni” is another word for condom in Italy). Whatever the origins of our word condom, and whatever you choose to call it, wear a condom. Condoms prevent unwanted pregnancies and prevent the spread of sexually transmitted diseases. vimax enlargement manhattan penis surgeon best penis enlarement surgery vigrx side effects best penis enlarement surgery vimax male penis enlargement pnis enlargement surgery vigrx ingredient home penis enlargement

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The concept of breast enlargement by ingesting pills is appealing to many women who are hesitant to undergo breast implant surgery. Many women view breast enlargement pills as an easy and inexpensive way of increasing their breast size. Breast enlargement pills contain phytoestrogens, a naturally occurring non-hormonal plant estrogen that promotes the growth of new breast tissue. These pills, as the manufacturers claim, activate the inactive estrogen sites in the breasts and increase breast size. The ingredients used in the pills are a combination of ancient herbs that naturally adjust the hormone levels and stimulate breast growth. Most of the pills contain seven ingredients, including fenugreek, don quai, saw palmetto, wild yam, blessed thistle, and damiana. The manufacturers assert that the consumers can see the result often in a couple of weeks and the consumption of pills for 3 – 6 months can lead to an increase in the breast by 1or 2 cup sizes. Do these pills really work? No, says some of the studies. On the contrary, they could be dangerous. Though manufacturers claim to have conducted research on their products, none of the medical journals have reportedly published their studies. It is understood that the studies, they claim, may have been derived from historical anecdotes or some isolated studies. The ingredients of breast enlargement pills, as per some studies, supposedly interfere with certain medicines. It is claimed that while fenugreek may interfere with medicines taken for blood clotting, chaste-berry tree may interfere with birth control pills. Don quai, one of the ingredients used in the pills is a known carcinogen. All breast enlargement pills are not the same, and therefore the results may differ for every individual. These pills are sold as herbal supplements and so the US Food and Drug Administration does not evaluate the product for their safety and effectiveness. Educating oneself about the ingredients found in the pills is of utmost importance. Individuals should also thoroughly go through the independent reviews of the products. Besides, individuals who take medication should equip themselves with the thorough knowledge about the potential negative effects of consuming the pills. vimax real penis enlargement compare penis enlargment pills top penis elargement pills male penis enlargment penis enlargement pill magna rx pennis enlargement excercises vimax easy enlargement free penis surgery way penis enlarement traction device penis enlagement technique

There are four areas of normal sexual function -- libido (desire), erection, ejaculation and orgasm. Problems in these areas can occur separately or in combination. Erectile dysfunction can often affect libido and ejaculation, especially when the erectile dysfunction persists. Each problem, regardless of cause is potentially treatable. When due to erectile dysfunction, they may resolve spontaneously when the erection problem is successfully treated, if not they need to be addressed separately. 1. Erectile dysfunction (ED) is a common problem. 2. ED is defined as the repeated inability to sustain an erection sufficient for sexual intercourse. 3. ED may be caused by physical factors, psychological factors or by medications. 4. ED may be caused by a problems in any of the components of the body that are required to produce an erection. These include: * Nerve impulses originating in the brain * Conduction of nerve impulses down the spinal cord * Conduction of nerve impulses between the spinal cord and the penis * Arteries and veins that supple the penis * The fibrous, muscular and vascular structures of the penis 5. Some common diseases are associated with an increased risk of erectile dysfunction. As some diseases progress, the disease will impact the function of nerves, blood vessels, vascular, and muscular structures of the penis. 6. Diseases associated with an increased risk of ED include diabetes, kidney disease, chronic alcoholism, vascular disease, multiple sclerosis, atherosclerosis. 7. In some cases, ED may be an early signal of heart or blood vessel disease. 8. Between 35 and 50 percent of men with diabetes experience ED. 9. Making an appointment with your physician or health care provider is the first step in the evaluation and treatment of ED. 10. ED can be treated! manual penile enlargment exercise penis enargement pic free penile enlargment elargement forum free matter penis size free exercise tip for pnis enlargement pennis enlargement traction device penis enargement pills penis girth enhancement penis enlagement technique

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penile enlargement device free penis enlagement tip pnis enlargement surgeries manual penile enlargment free exercise tip for penis enlagement free penis enhancement natural penile enlargement pills truth about penis enlargement pill penis enlagement technique

Babies take a lot of attention and a lot of love it is time well spent. The loving is always easy. Who could not love little babies? The attention and care for babies may present a problem for anyone who is not use to being around little one’s. Eventually you will get the hang of baby care and it is really not as complicated as you might think. Let’s start with the baby’s bottom changing a diaper is one of the first things that you will want to learn. For a little girl lift her legs with one hand and remove any poop with a wet washcloth. It is normally alright to use wet wipes as well. You can use a washcloth to clean the derriere the first few weeks to prevent any rash that a wet wipe might cause. To clean the genital area, wipe from the vagina toward the rectum. Dry the baby’s bottom with a soft cloth applies ointment around the genitals and on the buttocks to prevent diaper rash. One big difference for little boys is the penis. Make sure that you keep it covered or you might get sprayed. The procedure is basically the same make sure that he is cleaned, dried, and an ointment is applied around the genitals and buttocks. The next important thing for babies is feeding time. Breast feeding in the beginning is usually most desired. The baby eats and eats nature has done a pretty good job of providing you and your baby with the right equipment. At first you will find the nipples will be hard enough but they quickly get to be sore. Before you feed the baby we suggest that you get a heating pad or warm wet wash cloth warm nipples help the milk to flow much easier. After the feeding then we suggest that you use a cold pack to help you with the soreness. Most young mothers normally start within the third or fourth week giving the baby a bottle a day this helps the baby to get use to formula. We all want normal perfect babies but unfortunately birth defects can and do happen. It is important that all parents are aware of the most common birth defects and what can be done to prevent or treat your baby. Congenital heart defects are among the most common birth defects. It is said that about 25,000 U.S. babies are born with heart defects. These defects can be mild showing no symptoms at birth. The defect can cause baby’s ability to circulate oxygenated blood through the body. Today the prognosis for babies with congenital heart defects has improved significantly now it It is a good thing to do the corrections or treatment as soon as possible. The next birth defect is Cerebral Palsy. Cerebral Palsy baby’s movement is affected and so is the posture. It is caused by the part of the brain that controls muscle movement. Cerebral Palsy usually is not diagnosed until the child has reached the age of 2 or 3 years old. Two children out of 1.000 over the age of 3 have cerebral palsy. Currently in the United States, 500,000 individuals both children and adults are diagnosed with cerebral palsy. There is no cure for cerebral palsy but with treatment and physical therapy most children can significantly improve over time. Spina Bifida is the most common of a group of birth defects called neural tube defects. Spina Bifida affects approximately one in 2,000 babies. Scientists believe that baby’s who have spina bifida got it from their parents, however, they have found that most cases have been found in Hispanics mostly but there are cases in African Americans and Asians. Depending on the condition of the child treatment can range from none to several surgeries. Babies are truly a wonderful gift from God and my prayer has always been for my family that all the children are born happy and healthy. It is very unfortunate that sometimes this is always not true. I have a cousin who was born with cerebral palsy, a nephew who has spina bifida, and a brother who was born with multiple birth defects. These children have all added a special gift to our families. We have learned much better to love through their eyes. We all understand suffering through the eyes of a small child and grow stronger each day because of them. It would be most wonderful thing if all babies were born healthy but whatever happens please note your child is still special and will be loved by everyone.